Primary Graft Failure after Heart Transplantation
نویسندگان
چکیده
منابع مشابه
Primary Graft Failure after Heart Transplantation
Primary graft failure (PGF) is a devastating complication that occurs in the immediate postoperative period following heart transplantation. It manifests as severe ventricular dysfunction of the donor graft and carries significant mortality and morbidity. In the last decade, advances in pharmacological treatment and mechanical circulatory support have improved the outlook for heart transplant r...
متن کاملOutcome of extracorporeal membrane oxygenation for early primary graft failure after pediatric heart transplantation.
OBJECTIVES We sought to analyze the indications and outcome of extracorporeal membrane oxygenation (ECMO) for early primary graft failure and determine its impact on long-term graft function and rejection risk. BACKGROUND Early post-operative graft failure requiring ECMO can complicate heart transplantation. METHODS A retrospective review of all children requiring ECMO in the early period a...
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A 37-year-old man suffered from systolic heart failure as a result of idiopathic dilated cardiomyopathy since 1995 and was followed up in our cardiology department. In June 2006, the patient arrived at our outpatient clinic with an acute renal impairment and all manifestations of acute heart failure. He was already registered on the waiting list for heart transplantation (Eurotransplant) as a h...
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Primary graft dysfunction is a form of acute injury after lung transplantation that is associated with significant short- and long-term morbidity and mortality. Multiple mechanisms contribute to the pathogenesis of primary graft dysfunction, including ischemia reperfusion injury, epithelial cell death, endothelial cell dysfunction, innate immune activation, oxidative stress, and release of infl...
متن کاملEpstein-Barr virus associated graft failure following heart/lung transplantation.
A case is described of late pulmonary graft failure in a heart/lung transplant recipient. The major characteristics were alveolar fibrosis and a restrictive physiological deficit. Epstein-Barr virus was implicated as an aetiological agent using immunohistochemical analysis and by a response to treatment with ganciclovir.
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ژورنال
عنوان ژورنال: Journal of Transplantation
سال: 2011
ISSN: 2090-0007,2090-0015
DOI: 10.1155/2011/175768